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dc.contributor.authorKarandeni Dewage, Chinthani Shanika
dc.contributor.authorKlöppel, Coretta A.
dc.contributor.authorStotz, Henrik U.
dc.contributor.authorFitt, Bruce D. L.
dc.date.accessioned2019-10-16T00:06:29Z
dc.date.available2019-10-16T00:06:29Z
dc.date.issued2017
dc.identifier.citationKarandeni Dewage , C S , Klöppel , C A , Stotz , H U & Fitt , B D L 2017 , ' Host pathogen interactions in relation to management of light leaf spot disease (caused by Pyrenopeziza brassicae) on Brassica species ' , Crop and Pasture Science . https://doi.org/10.1071/CP16445
dc.identifier.issn1836-0947
dc.identifier.otherPURE: 11358621
dc.identifier.otherPURE UUID: 12824e0c-e88d-4ae4-984a-1592a2ccbd14
dc.identifier.otherScopus: 85040348095
dc.identifier.urihttp://hdl.handle.net/2299/21740
dc.description.abstractLight leaf spot, caused by Pyrenopeziza brassicae, is currently the most damaging disease problem in oilseed rape in the UK. According to recent survey data, the severity of epidemics has increased progressively across the UK, with current yield losses of up to £160M per annum in England and more severe epidemics in Scotland. Light leaf spot is a polycyclic disease with primary inoculum consisting of air-borne ascospores produced on diseased debris from the previous cropping season. Splash-dispersed conidia produced on diseased leaves are the main component of the secondary inoculum. P. brassicae is also able to infect and cause considerable yield losses on vegetable brassicas, especially Brussels sprouts. There may be spread of light leaf spot among different brassica species. Since they have a wide host range, Pyrenopeziza brassicae populations are likely to have considerable genetic diversity and there is evidence suggesting population variations between different regions, which need further study. Available disease-management tools are not sufficient to provide adequate control of the disease. There is a need to identify new sources of resistance, which can be integrated with fungicide applications to achieve sustainable management of light leaf spot. Several major resistance genes and quantitative trait loci have been identified in previous studies, but rapid improvements in the understanding of molecular mechanisms underpinning B. napus – P. brassicae interactions can be expected through exploitation of novel genetic and genomic information for brassicas and extracellular fungal pathogens.en
dc.language.isoeng
dc.relation.ispartofCrop and Pasture Science
dc.titleHost pathogen interactions in relation to management of light leaf spot disease (caused by Pyrenopeziza brassicae) on Brassica speciesen
dc.contributor.institutionSchool of Life and Medical Sciences
dc.contributor.institutionDepartment of Biological and Environmental Sciences
dc.contributor.institutionAgriculture, Food and Veterinary Sciences
dc.contributor.institutionGeography, Environment and Agriculture
dc.contributor.institutionCrop Protection and Climate Change
dc.contributor.institutionWeight and Obesity Research Group
dc.description.statusPeer reviewed
rioxxterms.versionVoR
rioxxterms.versionofrecordhttps://doi.org/10.1071/CP16445
rioxxterms.typeJournal Article/Review
herts.preservation.rarelyaccessedtrue


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