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dc.contributor.authorBaines, Simon D.
dc.contributor.authorSaxton, Katie
dc.contributor.authorFreeman, Jane
dc.contributor.authorWilcox, Mark H.
dc.date.accessioned2013-08-22T12:45:05Z
dc.date.available2013-08-22T12:45:05Z
dc.date.issued2006
dc.identifier.citationBaines , S D , Saxton , K , Freeman , J & Wilcox , M H 2006 , ' Tigecycline does not induce proliferation or cytotoxin production by epidemic Clostridium difficile strains in a human gut model ' , Journal of Antimicrobial Chemotherapy , vol. 58 , no. 5 , pp. 1062-5 . https://doi.org/10.1093/jac/dkl364
dc.identifier.issn0305-7453
dc.identifier.urihttp://hdl.handle.net/2299/11456
dc.description.abstractData on the risk of Clostridium difficile infection (CDI) associated with specific antibiotics are difficult to obtain because of confounding clinical factors. It is particularly important to evaluate the propensity of new antibiotics to induce CDI. We have examined the propensity of tigecycline to induce CDI using a human gut model.en
dc.format.extent4
dc.format.extent121698
dc.language.isoeng
dc.relation.ispartofJournal of Antimicrobial Chemotherapy
dc.titleTigecycline does not induce proliferation or cytotoxin production by epidemic Clostridium difficile strains in a human gut modelen
dc.contributor.institutionSchool of Life and Medical Sciences
dc.contributor.institutionMicrobiology and Biotechnology
dc.contributor.institutionBiosciences Research Group
dc.contributor.institutionCentre for Research in Mechanisms of Disease and Drug Discovery
dc.contributor.institutionDepartment of Clinical, Pharmaceutical and Biological Science
dc.contributor.institutionCentre for Future Societies Research
dc.description.statusPeer reviewed
rioxxterms.versionofrecord10.1093/jac/dkl364
rioxxterms.typeJournal Article/Review
herts.preservation.rarelyaccessedtrue


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